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with endothelial function in the coronary arteries and has been rmly established as a coronary risk factor Hoch et al (2003) and Rickenlund et al (2005) showed that amenorrheic female athletes display a signi cantly greater degree of endothelial dysfunction is than do their eumenorrheic peers, as evidenced by impaired FMD Furthermore, each group demonstrated reversal of these abnormalities with therapeutic intervention (Hoch et al 2003; Rickenlund et al 2005) The Hoch group used an educational intervention that resulted in the resumption of normal menstrual function in seven of nine subjects These seven subjects showed signi cant improvement in their FMD The Rickenlund group used oral contraceptives for 9 months, and their amenorrheic athletes also showed signi cant improvements in their FMD.

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While research in this area remains young, the initial data indicate that young athletes with hypothalamic amenorrhea may face increased cardiovascular risk, despite their highly active lifestyles, and that this risk is potentially improved with restoration of energy balance or estrogen supplementation Clinical evaluation of the female athlete with amenorrhea The clinical evaluation of a female athlete with amenorrhea begins with an understanding of the differential diagnosis of amenorrhea Too often, the assumption is made that the woman s amenorrhea is coming from her athletic participation without appropriate investigation into other potential causes Too often, as well, the patient is told that she is experiencing a benign phenomenon about which she need not worry The starting point for evaluating a female athlete with amenorrhea is with a differential diagnosis for both primary and secondary amenorrhea that includes the hypothalamic amenorrhea discussed above, but also goes beyond it to other causes.

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A careful history and physical examination followed by a focused laboratory examination is also essential Reindollar et al (1989) broke down the differential diagnosis of primary amenorrhea into three broad categories: hypergonadotrophic hypogonadism, hypogonadotrophic hypogonadism, and eugonadism..

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De nition and epidemiology of sudden cardiac death Sudden cardiac death (SCD) is de ned as a witnessed or unwitnessed natural death resulting from sudden cardiac arrest occurring unexpectedly within 6 hours of a previously normal state of health Vigorous exertion acutely increases the risk of sudden death in individuals with known or occult cardiac disease, but the absolute cardiac risk of exercise is extremely low The annual incidence of sudden death among high school and college athletes ranges between 1 in 100,000 and 1 in 300,000 (Maron et al 1998a; Van Camp et al 1995) The incidence is signi cantly higher in males than females (1 in 133,000 in men and 1 in 769,000 in women; Van Camp et al 1995).

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